![]() Upon scheduled wake-up time, subjective sleep quality and caffeine withdrawal symptoms were assessed. After 9 days of continuous treatment, electroencephalographically (EEG)-derived sleep structure and intensity were recorded during a scheduled 8-h nighttime sleep episode starting 8 (caffeine condition) and 15 h (withdrawal condition) after the last caffeine intake. ![]() To address this question, we investigated the sleep of twenty male young habitual caffeine consumers during a double-blind, randomized, crossover study including three 10-day conditions: caffeine (3 × 150 mg caffeine daily), withdrawal (3 × 150 mg caffeine for 8 days, then switch to placebo), and placebo (3 × placebo daily). However, it is not clear whether these sleep disturbances disappear when caffeine is continuously consumed during daytime, which is common for most coffee drinkers. 2010 121:1904–1911.Acute caffeine intake can delay sleep initiation and reduce sleep intensity, particularly when consumed in the evening. Excessive supraventricular ectopic activity and increased risk of atrial fibrillation and stroke. ![]() 2013 159:721–728.īinici Z, Intzilakis T, Nielsen OW, Kober L, Sajadieh A. Atrial ectopy as a predictor of incident atrial fibrillation: a cohort study. 1998 339:659–666.ĭewland TA, Vittinghoff E, Mandyam MC, Heckbert SR, Siscovick DS, Stein PK, Psaty BM, Sotoodehnia N, Gottdiener JS, Marcus GM. Spontaneous initiation of atrial fibrillation by ectopic beats originating in the pulmonary veins. Haissaguerre M, Jais P, Shah DC, Takahashi A, Hocini M, Quiniou G, Garrigue S, Le Mouroux A, Le Metayer P, Clementy J. Prevalence of premature ventricular contractions in a population of African American and white men and women: the Atherosclerosis Risk in Communities (ARIC) study. Simpson RJ Jr, Cascio WE, Schreiner PJ, Crow RS, Rautaharju PM, Heiss G. Cardiac arrhythmias on 24‐h ambulatory electrocardiography in older women and men: the Cardiovascular Health Study. Manolio TA, Furberg CD, Rautaharju PM, Siscovick D, Newman AB, Borhani NO, Gardin JM, Tabatznik B. Published on behalf of the American Heart Association, Inc., by Wiley Blackwell. In the largest study to evaluate dietary patterns and quantify cardiac ectopy using 24-hour Holter monitoring, we found no relationship between chronic consumption of caffeinated products and ectopy.Īrrhythmia diet electrophysiology epidemiology. In examining combined dietary intake of coffee, tea, and chocolate as a continuous measure, no relationships were observed after multivariable adjustment: 0.48% fewer PACs/h (95% CI -4.60 to 3.64) and 2.87% fewer PVCs/h (95% CI -8.18 to 2.43) per 1-serving/week increase in consumption. After adjustment for potential confounders, more frequent consumption of these products was not associated with ectopy. ![]() There were no differences in the number of PACs or PVCs/h across levels of coffee, tea, and chocolate consumption. The median numbers of PACs and PVCs/h and interquartile ranges were 3 (1-12) and 1 (0-7), respectively. Among 1388 participants (46% male, mean age 72 years), 840 (61%) consumed ≥1 caffeinated product per day. The main outcomes were PACs/h and PVCs/hour. Frequencies of habitual coffee, tea, and chocolate consumption were assessed using a picture-sort food frequency survey. We studied Cardiovascular Health Study participants with a baseline food frequency assessment, 24-hour ambulatory electrocardiography (Holter) monitoring, and without persistent atrial fibrillation. As certain caffeinated products may have cardiovascular benefits, recommendations against them may be detrimental. Though experts associate premature atrial contractions (PACs) and premature ventricular contractions (PVCs) with caffeine, there are no data to support this relationship in the general population. Premature cardiac contractions are associated with increased morbidity and mortality.
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